LogFAQs > #948072389

I won't be getting the COVID vaccine, and I feel justified after reading official documentation from the FDA, uk.gov, as well as research papers linked. I have a few questions I'm trying to put my mind at ease about.

I'm concerned that there were no animal studies, and more specifically, the challenge phase of animal studies. Typically, animals receive the vaccine, then it is challenged by receiving the real virus. This is important, because the SAR-COV-1 vaccine failed, and also coronavirus vaccines tested in cats have failed after the the challenge, resulting in deaths of the cats.

See: https://pubmed.ncbi.nlm.nih.gov/2154621/ (paper on the challenges of vaccinating cats with a coronavirus vaccine, resulting in death of the cats)

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7115537/

Antibody-dependent enhancement (ADE) has been observed in vaccinated and wild-type infections of FIP. ADE is thought to potentiate viral infection through the infection of macrophages. Viral entry into macrophages occurs when antibodies bind the virus and attach to macrophages via the Fc region of the antibody and its interaction with cell surface expressed Fc receptors [46]. Neutralizing antibodies can also be enhancing antibodies if antibody titer is low or is of the IgG class [47], [48]. Because macrophages increase with viral disease, this cell type may provide an abundant reservoir for the virus and thus expansion of the virus in the host. Some similarities between FIP and SARS exist. First, in both cases macrophages can be infected with the virus [9], [49], and in the case of SARS, the etiology of disease is contributed by infiltrating alveolar macrophages leading to pneumonitis [8]. Second, the treatment with corticosteroids and/or interferon alpha ameliorates SARS disease [50], suggesting an inflammatory, immune-mediated disease. While there has been no observation of ADE during SARS infection, it is worth noting that one coronavirus, FIPV, is capable of eliciting ADE and in the evaluation of vaccines, we may want to consider this possible outcome. However, the difficulty in testing animal models for ADE bears the caveat that if ADE is not observed; it has not proved that vaccines are safe with regard to ADE in humans. In contrast, if an animal model for ADE is developed, we may learn more about the mechanism of SARS-induced ADE, which may help form the basis for developing guidelines for safe vaccine development.

I'm concerned about an ADE (Antibody Dependent Enhancement). And also mid to long term effects of the vaccine. Does having the spike proteins from the vaccine put someone at risk for problems down the road for things like cancer? Does this vaccine create issues with fertility?

See the FDA's document for side effects of the they are watching for. On page 16, Vaccine enhanced disease is listed and so is pregnancy issues. There's also a large list of severe illnesses and autoimmune conditions listed.

https://www.fda.gov/media/143557/download?fbclid=IwAR2V6aqOmjD2WZ2uc3VQEUrDDEKDkpHGMAPXkYlb1U9wgZqu7AOp81DsyBE

Note that Transverse myelitis is listed, which is a spinal and neurological condition, which occurred in the Astrazenica/Oxford vaccine

We also know from UK documentation of the vaccine, that there is no data on fertility. Please see this file on page 6:

https://assets.publishing.service.gov.uk/government/uploads/system/uploads/attachment_data/file/941452/Information_for_healthcare_professionals.pdf?fbclid=IwAR1DdnB-VVphxOKrmXmi0iAOXZOTeNBfydQPA4s4_QmN4gQXID5gKrT5iQE

"Fertility - It is unknown whether COVID-19 mRNA Vaccine BNT162b2 has an impact on fertility"

Don't you think we should know this before taking the vaccine?

Anyways, these are my concerns, looking for some answers. Any thoughts?

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